Allograft dysfunction and parenchymal necrosis associated with renal artery stenosis and perigraft hematoma after kidney transplantation

Bibliographic Details
Title: Allograft dysfunction and parenchymal necrosis associated with renal artery stenosis and perigraft hematoma after kidney transplantation
Authors: Han Sae Kim, Jin Ho Lee, Dong Yeol Lee, Hee Yeoun Kim, Dong Han Kim, Joon Seok Oh, Yong Hun Sin, Joong Kyung Kim, Seun Deuk Hwang
Source: Korean Journal of Transplantation, Vol 34, Iss 2, Pp 126-131 (2020)
Publisher Information: Korean Society for Transplantation, 2020.
Publication Year: 2020
Collection: LCC:Medical technology
Subject Terms: kidney transplantation, transplant renal artery stenosis, graft dysfunction, Medical technology, R855-855.5
More Details: Transplant renal artery stenosis (TRAS) is one cause of allograft dysfunction. TRAS causes parenchymal necrosis and graft insufficiency. Herein, we report the case of a 40-year-old female with end-stage renal disease due to immunoglobulin A nephropathy, who underwent kidney transplantation with her elder sister. The surgery was successful and the allograft showed primary graft function. At postoperative day (POD) 2, urine output decreased sharply. We checked a non-enhanced abdominal computed tomography scan which showed subcapsular and pelvic cavity hematomas. She underwent hematoma removal surgery with renal upper polar capsulotomy. Bleeding control was successful, but her serum creatinine was 5.4 mg/dL. At POD 25, abdomen magnetic resonance angiography showed significant stenosis at the anastomosis site between the graft renal artery and the recipient’s internal iliac artery. Then, percutaneous transluminal angioplasty was implemented. Significant stenosis (>80%) was detected at the anastomotic site and a 5-mm stent was inserted at stenotic lesion with post-stent balloon angioplasty using a 5-mm balloon catheter. The renal arterial diameter and blood flow were normalized. At postoperative 5 months, a 99mTc dimercaptosuccinic acid scan showed multiple focal radioisotope defects. At 54 months after renal transplantation, her serum creatinine level was 4.0 mg/dL and her glomerular filtration rate was 13 mL/min/1.73 m2. Hence, we report that TRAS can cause parenchymal necrosis and allograft dysfunction.
Document Type: article
File Description: electronic resource
Language: English
ISSN: 2671-8790
Relation: http://journaleditor.inforang.com/journal/view.html?doi=10.4285/kjt.2020.34.2.126; https://doaj.org/toc/2671-8790
DOI: 10.4285/kjt.2020.34.2.126
Access URL: https://doaj.org/article/b274d37b11c742668e139bc930cbac15
Accession Number: edsdoj.b274d37b11c742668e139bc930cbac15
Database: Directory of Open Access Journals
More Details
ISSN:26718790
DOI:10.4285/kjt.2020.34.2.126
Published in:Korean Journal of Transplantation
Language:English