Tuberculosis: New Aspects of an Old Disease

Bibliographic Details
Title: Tuberculosis: New Aspects of an Old Disease
Authors: Luisa Jordao, Otilia V. Vieira
Source: International Journal of Cell Biology, Vol 2011 (2011)
Publisher Information: Hindawi Limited, 2011.
Publication Year: 2011
Collection: LCC:Cytology
Subject Terms: Cytology, QH573-671
More Details: Tuberculosis is an ancient infectious disease that remains a threat for public health around the world. Although the etiological agent as well as tuberculosis pathogenesis is well known, the molecular mechanisms underlying the host defense to the bacilli remain elusive. In this paper we focus on the innate immunity of this disease reviewing well-established and consensual mechanisms like Mycobacterium tuberculosis interference with phagosome maturation, less consensual mechanism like nitric oxide production, and new mechanisms, such as mycobacteria translocation to the cytosol, autophagy, and apoptosis/necrosis proposed mainly during the last decade.
Document Type: article
File Description: electronic resource
Language: English
ISSN: 1687-8876
1687-8884
Relation: https://doaj.org/toc/1687-8876; https://doaj.org/toc/1687-8884
DOI: 10.1155/2011/403623
Access URL: https://doaj.org/article/2fe8f89374424a0280bde524cd38f8b8
Accession Number: edsdoj.2fe8f89374424a0280bde524cd38f8b8
Database: Directory of Open Access Journals
FullText Links:
  – Type: pdflink
    Url: https://content.ebscohost.com/cds/retrieve?content=AQICAHjPtM4BHU3ZchRwgzYmadcigk49r9CVlbU7V5F6lgH7WwHgi9t8MbJfQ2nrnDGkgAWTAAAA4TCB3gYJKoZIhvcNAQcGoIHQMIHNAgEAMIHHBgkqhkiG9w0BBwEwHgYJYIZIAWUDBAEuMBEEDN6acCcyPZd_5khvjgIBEICBmeTcudaEt4D6aCfKGK3ZBMCnzgLD4VLUPYLM0Zw_duWei-2W8kvwToAJe992DgjHVRNyNcZ7o8JVg4XrdKp0FcUwu0HRusJOg_sFGJWKMrrRY-mBFnIrcHb_SA6CQ2_sdPD9nkSpOkhLsZPcooLLbQra0IdlrdaM3gExEbxnipSjTMm9zzkwvrbmbtZ6TEQ4AoY1eGnxXZIBvA==
Text:
  Availability: 0
CustomLinks:
  – Url: https://resolver.ebsco.com/c/xy5jbn/result?sid=EBSCO:edsdoj&genre=article&issn=16878876&ISBN=&volume=2011&issue=&date=20110101&spage=&pages=&title=International Journal of Cell Biology&atitle=Tuberculosis%3A%20New%20Aspects%20of%20an%20Old%20Disease&aulast=Luisa%20Jordao&id=DOI:10.1155/2011/403623
    Name: Full Text Finder (for New FTF UI) (s8985755)
    Category: fullText
    Text: Find It @ SCU Libraries
    MouseOverText: Find It @ SCU Libraries
  – Url: https://doaj.org/article/2fe8f89374424a0280bde524cd38f8b8
    Name: EDS - DOAJ (s8985755)
    Category: fullText
    Text: View record from DOAJ
    MouseOverText: View record from DOAJ
Header DbId: edsdoj
DbLabel: Directory of Open Access Journals
An: edsdoj.2fe8f89374424a0280bde524cd38f8b8
RelevancyScore: 843
AccessLevel: 3
PubType: Academic Journal
PubTypeId: academicJournal
PreciseRelevancyScore: 842.654235839844
IllustrationInfo
Items – Name: Title
  Label: Title
  Group: Ti
  Data: Tuberculosis: New Aspects of an Old Disease
– Name: Author
  Label: Authors
  Group: Au
  Data: <searchLink fieldCode="AR" term="%22Luisa+Jordao%22">Luisa Jordao</searchLink><br /><searchLink fieldCode="AR" term="%22Otilia+V%2E+Vieira%22">Otilia V. Vieira</searchLink>
– Name: TitleSource
  Label: Source
  Group: Src
  Data: International Journal of Cell Biology, Vol 2011 (2011)
– Name: Publisher
  Label: Publisher Information
  Group: PubInfo
  Data: Hindawi Limited, 2011.
– Name: DatePubCY
  Label: Publication Year
  Group: Date
  Data: 2011
– Name: Subset
  Label: Collection
  Group: HoldingsInfo
  Data: LCC:Cytology
– Name: Subject
  Label: Subject Terms
  Group: Su
  Data: <searchLink fieldCode="DE" term="%22Cytology%22">Cytology</searchLink><br /><searchLink fieldCode="DE" term="%22QH573-671%22">QH573-671</searchLink>
– Name: Abstract
  Label: Description
  Group: Ab
  Data: Tuberculosis is an ancient infectious disease that remains a threat for public health around the world. Although the etiological agent as well as tuberculosis pathogenesis is well known, the molecular mechanisms underlying the host defense to the bacilli remain elusive. In this paper we focus on the innate immunity of this disease reviewing well-established and consensual mechanisms like Mycobacterium tuberculosis interference with phagosome maturation, less consensual mechanism like nitric oxide production, and new mechanisms, such as mycobacteria translocation to the cytosol, autophagy, and apoptosis/necrosis proposed mainly during the last decade.
– Name: TypeDocument
  Label: Document Type
  Group: TypDoc
  Data: article
– Name: Format
  Label: File Description
  Group: SrcInfo
  Data: electronic resource
– Name: Language
  Label: Language
  Group: Lang
  Data: English
– Name: ISSN
  Label: ISSN
  Group: ISSN
  Data: 1687-8876<br />1687-8884
– Name: NoteTitleSource
  Label: Relation
  Group: SrcInfo
  Data: https://doaj.org/toc/1687-8876; https://doaj.org/toc/1687-8884
– Name: DOI
  Label: DOI
  Group: ID
  Data: 10.1155/2011/403623
– Name: URL
  Label: Access URL
  Group: URL
  Data: <link linkTarget="URL" linkTerm="https://doaj.org/article/2fe8f89374424a0280bde524cd38f8b8" linkWindow="_blank">https://doaj.org/article/2fe8f89374424a0280bde524cd38f8b8</link>
– Name: AN
  Label: Accession Number
  Group: ID
  Data: edsdoj.2fe8f89374424a0280bde524cd38f8b8
PLink https://login.libproxy.scu.edu/login?url=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsdoj&AN=edsdoj.2fe8f89374424a0280bde524cd38f8b8
RecordInfo BibRecord:
  BibEntity:
    Identifiers:
      – Type: doi
        Value: 10.1155/2011/403623
    Languages:
      – Text: English
    Subjects:
      – SubjectFull: Cytology
        Type: general
      – SubjectFull: QH573-671
        Type: general
    Titles:
      – TitleFull: Tuberculosis: New Aspects of an Old Disease
        Type: main
  BibRelationships:
    HasContributorRelationships:
      – PersonEntity:
          Name:
            NameFull: Luisa Jordao
      – PersonEntity:
          Name:
            NameFull: Otilia V. Vieira
    IsPartOfRelationships:
      – BibEntity:
          Dates:
            – D: 01
              M: 01
              Type: published
              Y: 2011
          Identifiers:
            – Type: issn-print
              Value: 16878876
            – Type: issn-print
              Value: 16878884
          Numbering:
            – Type: volume
              Value: 2011
          Titles:
            – TitleFull: International Journal of Cell Biology
              Type: main
ResultId 1