The Role of Toll-Like Receptors (TLRs) in Infection and Inflammation 2.0

Contributors: Kircheis, Ralf, Planz, Oliver

Subject Terms: Toll-like receptor, COVID-19, SARS-CoV-2, innate immunity, host genetics, MYD88, CCL4, CCL20, CCR2, atherosclerosis, statin, toll-like receptor, TLR agonist, HIV-1, co-infection, HBV, HCV, Toll-like receptor 2, dimerization, homodimerization, heterodimerization, inflammation, neuroinflammation, neurodegeneration, TLRs, TLR-4, lethal COVID-19, lung disease, macrophage shift, ARDS, CD68, galectin-3, CD163, M1 macrophages, M2 macrophages, immune-to-brain communication, lung–brain axis, lipopolysaccharide, acute respiratory distress syndrome, cytokines, lipid mediators, omega-3 fatty acids, resolvin E1 receptors, TNFα, cfDNA, TLR (toll-like receptor) 9, cathelicidin anti-microbial peptide, adipocyte, adipose tissue, Toll-like receptors, immunity, adaptive immunity, ligands, toll-like receptor (TLR), myeloid differentiation factor-2 (MD-2), TLR4/MD-2, Omicron, spike protein, cytokine storm, NF-kappa B, TLR, brain, neurogenesis, blood–brain barrier, behavior, cognition, TLR7, NOD2, synergy, type I IFN, experimental autoimmune encephalomyelitis, complement, C5a, sepsis, heart dysfunction, arrhythmia, cardiomyocytes, neutrophils, Research and information: general, Biology, life sciences

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Access URL: https://directory.doabooks.org/handle/20.500.12854/152864
https://mdpi.com/books/pdfview/book/10053