Functional monovalency amplifies the pathogenicity of anti-MuSK IgG4 in myasthenia gravis

Authors: Vergoossen, Dana L. E., Plomp, Jaap J., Gstöttner, Christoph, Fillié-Grijpma, Yvonne E., Augustinus, Roy, Verpalen, Robyn, Wuhrer, Manfred, Parren, Paul W. H. I., Dominguez-Vega, Elena, van der Maarel, Silvère M., Verschuuren, Jan J., Huijbers, Maartje G.

Source: Proceedings of the National Academy of Sciences of the United States of America, 2021 Mar . 118(13), 1-11.

Access URL: https://www.jstor.org/stable/27039797

Change of voltage‐gated sodium channel repertoire in skeletal muscle of a MuSK myasthenia gravis mouse model.

Authors: Butenko, Olena, Jensen, Stine Marie, Fillié‐Grijpma, Yvonne E., Verpalen, Robyn, Verschuuren, Jan J., van der Maarel, Silvère M., Huijbers, Maartje G., Plomp, Jaap J.

Source: European Journal of Neuroscience; Jun2024, Vol. 59 Issue 12, p3292-3308, 17p

Subject Terms: SODIUM channels, ACTION potentials, LABORATORY mice, MUSCLE weakness, ANIMAL disease models, MYASTHENIA gravis, SKELETAL muscle