TAK-242 improves sepsis-associated acute kidney injury in rats by inhibiting the TLR4/NF-κB signaling pathway

Authors: Yan-mei Xia, Yu-qian Guan, Ji-fang Liang, Wei-dong Wu

Source: Renal Failure, Vol 46, Iss 1 (2024)

Subject Terms: Sepsis-associated acute kidney injury (SA-AKI), TAK-242, oxidative stress, mitochondrial dysfunction, inflammatory factors, toll-like receptor 4 (TLR4)/nuclear factor κB (NF-κB) signaling pathway, Diseases of the genitourinary system. Urology, RC870-923

File Description: electronic resource

Relation: https://doaj.org/toc/0886-022X; https://doaj.org/toc/1525-6049

Access URL: https://doaj.org/article/535110157df24377afd50559a4bcfc3e

The role of inflammatory response and metabolic reprogramming in sepsis-associated acute kidney injury: mechanistic insights and therapeutic potential

Authors: An-Bu Liu, Bin Tan, Ping Yang, Na Tian, Jin-Kui Li, Si-Cong Wang, Li-Shan Yang, Lei Ma, Jun-Fei Zhang

Source: Frontiers in Immunology, Vol 15 (2024)

Subject Terms: sepsis associated acute kidney injury (SA-AKI), inflammatory response, metabolic reprogramming, regulatory cell death (RCD), mechanism, Immunologic diseases. Allergy, RC581-607

File Description: electronic resource

Relation: https://www.frontiersin.org/articles/10.3389/fimmu.2024.1487576/full; https://doaj.org/toc/1664-3224

Access URL: https://doaj.org/article/de1226da61454df9bf4a5cbe856d7973