Synchronisation of apical constriction and cell cycle progression is a conserved behaviour of pseudostratified neuroepithelia informed by their tissue geometry

Authors: Ampartzidis, Ioakeim, Efstathiou, Christoforos, Paonessa, Francesco, Thompson, Elliott M., Wilson, Tyler, McCann, Conor J., Greene, Nicholas DE., Copp, Andrew J., Livesey, Frederick J., Elvassore, Nicola, Giobbe, Giovanni G., De Coppi, Paolo, Maniou, Eirini, Galea, Gabriel L.

Source: Developmental biology. 494:60-70

Availability: http://explore.bl.uk/primo_library/libweb/action/display.do?tabs=detailsTab&gathStatTab=true&ct=display&fn=search&doc=ETOCvdc_100179165663.0x000001&indx=1&recIds=ETOCvdc_100179165663.0x000001

Microtubules Deform the Nuclear Membrane and Disrupt Nucleocytoplasmic Transport in Tau-Mediated Frontotemporal Dementia

Authors: Paonessa, Francesco, Evans, Lewis D., Solanki, Ravi, Larrieu, Delphine, Wray, Selina, Hardy, John, Jackson, Stephen P., Livesey, Frederick J.

Source: In Cell Reports 15 January 2019 26(3):582-593

Regulation of neural gene transcription by optogenetic inhibition of the RE1-silencing transcription factor

Authors: Paonessa, Francesco, Criscuolo, Stefania, Sacchetti, Silvio, Amoroso, Davide, Scarongella, Helena, Bisogni, Federico Pecoraro, Carminati, Emanuele, Pruzzo, Giacomo, Maragliano, Luca, Cesca, Fabrizia, Benfenati, Fabio

Source: Proceedings of the National Academy of Sciences of the United States of America, 2016 Jan 01. 113(1), E91-E100.

Access URL: https://www.jstor.org/stable/26467332

Specificity Protein 1 (Sp1)-dependent Activation of the Synapsin I Gene (SYN1) Is Modulated by RE1-silencing Transcription Factor (REST) and 5′-Cytosine-Phosphoguanine (CpG) Methylation

Authors: Paonessa, Francesco, Latifi, Shahrzad, Scarongella, Helena, Cesca, Fabrizia, Benfenati, Fabio

Source: In Journal of Biological Chemistry 1 February 2013 288(5):3227-3239

Functional Variants of the HMGAI Gene and Type 2 Diabetes Mellitus.

Authors: Chiefari, Eusebio, Tanyolaç, Sinan, Paonessa, Francesco

Source: JAMA: Journal of the American Medical Association; March 2 2011, Vol. 305 Issue 9, p903-912, 10p

The cAMP-HMGA1-RBP4 system: a novel biochemical pathway for modulating glucose homeostasis

Authors: Foti Daniela, Colantuoni Vittorio, Brunetti Giuseppe, Lupo Angelo, Palmieri Dario, Le Pera Ilaria, Iiritano Stefania, Paonessa Francesco, Chiefari Eusebio, Gulletta Elio, De Sarro Giovambattista, Fusco Alfredo, Brunetti Antonio

Source: BMC Biology, Vol 7, Iss 1, p 24 (2009)

Subject Terms: Biology (General), QH301-705.5

File Description: electronic resource

Relation: http://www.biomedcentral.com/1741-7007/7/24; https://doaj.org/toc/1741-7007

Access URL: https://doaj.org/article/87a6b563f8e04b7299d176d324a0757e

New Target Genes for the Peroxisome Proliferator-Activated Receptor-γ (PPARγ) Antitumour Activity: Perspectives from the Insulin Receptor.

Authors: Foti, Daniela P.^1,2, Paonessa, Francesco¹, Chiefari, Eusebio¹, Brunetti, Antonio^1,3

Source: PPAR Research. 2009, Special section p1-8. 8p. 2 Diagrams.

Subject Terms: *INSULIN receptors, *NUCLEAR receptors (Biochemistry), *PEROXISOMES, *ANTINEOPLASTIC agents, *METABOLISM

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The cAMP-HMGA1-RBP4 system: a novel biochemical pathway for modulating glucose homeostasis.

Authors: Chiefari, Eusebio, Paonessa, Francesco, Iiritano, Stefania, Le Pera, Ilaria, Palmieri, Dario, Brunetti, Giuseppe, Lupo, Angelo, Colantuoni, Vittorio, Foti, Daniela, Gulletta, Elio, De Sarro, Giovambattista, Fusco, Alfredo, Brunetti, Antonio

Source: BMC Biology; 2009, Vol. 7, Special section p1-13, 13p, 9 Graphs

Subject Terms: GLUCOSE, INSULIN, HOMEOSTASIS, PHENOTYPES, BIOCHEMISTRY

Lack of the architectural factor HMGA1 causes insulin resistance and diabetes in humans and mice.

Authors: Foti, Daniela, Chiefari, Eusebio, Fedele, Monica, Iuliano, Rodolfo, Brunetti, Leonardo, Paonessa, Francesco, Manfioletti, Guidalberto, Barbetti, Fabrizio, Brunetti, Arturo, Croce, Carlo M., Fusco, Alfredo, Brunetti, Antonio

Source: Nature Medicine; Jul2005, Vol. 11 Issue 7, p765-773, 9p

Subject Terms: TYPE 2 diabetes, INSULIN resistance, PROTEINS, GENETIC transcription, INSULIN receptors

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